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Eosinophilic asthma exacerbations may be clinically more severe than non-eosinophilic exacerbation
Type 2 inflammation is present in patients during virus-induced asthma exacerbations, to the same degree as non-viral exacerbations
PLA2R1 is increased in the airway epithelium in asthma, and serves as a regulator of airway hyperresponsiveness, airway permeability, antigen sensitization, and airway inflammation
Human rhinovirus infection delays repair and inhibits apoptotic processes in epithelial cells from non-asthmatic and asthmatic children
Genetic factors in airway epithelial cells that are functionally associated with asthma pathogenesis
The Respiratory Environmental Health team conducts research in early life determinants of lung growth and development, respiratory environmental health, and mechanisms of airway dysfunction in asthma and other respiratory disease.
The Foundations of Lung Disease Team is focused on improving the diagnosis, treatment, and lifelong care of childhood lung disease.
We’ve heard from families that trustworthy information about preterm-associated lung disease is difficult to find. In response, we’ve created resources to empower families with the knowledge they need to manage these challenges.
This project investigates how different populations of cells within the respiratory tract immune system are altered during a viral infection.
This study is designed to identify the specific unique immune cell response that occurs in these children with recurrent disease.